2-way analysis of variance (anova) Search Results


statistical 2 way anova analysis  (S2 Statistical Solutions)
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S2 Statistical Solutions statistical 2 way anova analysis
Statistical 2 Way Anova Analysis, supplied by S2 Statistical Solutions, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way anova analysis, t-test, or nonparametric test  (GraphPad Software Inc)
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GraphPad Software Inc 2-way anova analysis, t-test, or nonparametric test
2 Way Anova Analysis, T Test, Or Nonparametric Test, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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two-tailed 2-way analysis of variance (anova) test  (GraphPad Software Inc)
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Two Tailed 2 Way Analysis Of Variance (Anova) Test, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way repeated measures in 1 dimension (time) analysis of variance (anova)  (GraphPad Software Inc)
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2-way mixed analysis of variance (anova)  (RStudio)
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RStudio 2-way mixed analysis of variance (anova)
2 Way Mixed Analysis Of Variance (Anova), supplied by RStudio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way repeated-measures anova and the post hoc analysis based on dunnett  (SAS institute)
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SAS institute 2-way repeated-measures anova and the post hoc analysis based on dunnett
Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on <t>Dunnett</t> were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.
2 Way Repeated Measures Anova And The Post Hoc Analysis Based On Dunnett, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way anova and scheffé post-hoc analysis sas/stat version 6  (SAS institute)
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SAS institute 2-way anova and scheffé post-hoc analysis sas/stat version 6
Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on <t>Dunnett</t> were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.
2 Way Anova And Scheffé Post Hoc Analysis Sas/Stat Version 6, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way anova using the scheffe f-test for post hoc analysis  (Jandel Engineering)
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Jandel Engineering 2-way anova using the scheffe f-test for post hoc analysis
Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on <t>Dunnett</t> were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.
2 Way Anova Using The Scheffe F Test For Post Hoc Analysis, supplied by Jandel Engineering, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way anova with tukey or bonferroni post-hoc analysis graphpad prism  (GraphPad Software Inc)
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GraphPad Software Inc 2-way anova with tukey or bonferroni post-hoc analysis graphpad prism
Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on <t>Dunnett</t> were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.
2 Way Anova With Tukey Or Bonferroni Post Hoc Analysis Graphpad Prism, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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2-way analysis of variance (anova)  (Dynamic Microsystems Inc)
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Dynamic Microsystems Inc 2-way analysis of variance (anova)
Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on <t>Dunnett</t> were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.
2 Way Analysis Of Variance (Anova), supplied by Dynamic Microsystems Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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repeated measures two-way analysis of variance  (SYSTAT)
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SYSTAT repeated measures two-way analysis of variance
Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on <t>Dunnett</t> were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.
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Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on Dunnett were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.

Journal: Journal of neurophysiology

Article Title: Pharmacology of Nicotinic Receptors in PreBötzinger Complex That Mediate Modulation of Respiratory Pattern

doi:

Figure Lengend Snippet: Systems level effects of Nic and Antag. A: Nic (0.5 μM) increased respiratory frequency; this effect slightly desensitized. Nic was bath applied at time 0 min and was continuously present for 30 min (n = 3). B: the effects of nicotinic antagonists α-BgTx (n = 4) or MLA (n = 6) on Nic-induced increase in respiratory frequency are not significant compared with control (Ctrl). DH-β-E (n = 4), HMT (n = 5) and d-TC (n = 6) reversed these effects. Control value was taken at about 10 min during bath application of nicotine in the group of control experiment as A without adding antagonist. Ten minutes was equivalent to the time the effects of antagonists were measured (4–6 min after Antag application and Antag was applied 3–5 min after Nic application) in the groups of antagonist experiments. Two-way repeated-measures ANOVA and the post hoc analysis based on Dunnett were used to analyze the change in frequency with antagonists compared with the control desensitization. C: the effects of α-BgTx or MLA on nicotine-induced decrease in amplitude of inspiratory bursts were not significant, while DH-β-E, HMT and d-TC reversed these nicotinic effects. One-way repeated-measures ANOVA for each group and post hoc analysis based on Tukey (SAS Institute 1999) were used. *, statistically significant. The frequency and amplitude data were normalized to baseline values prior to Nic application for each preparation. Raw data (nonnormalized) were used in statistical tests.

Article Snippet: The decrease in frequency with DH- β -E, HMT, and d -TC were significant compared with the control desensitization while the decrease with α -BgTx or MLA was not (analyzed by 2-way repeated-measures ANOVA and the post hoc analysis based on Dunnett ( SAS Institute, 1999 ).

Techniques: Control